Effects of oral propranolol on parathyroid hormone secretion in chronic renal failure.
نویسندگان
چکیده
differences show that delivery by lower segment caesarean section is more dangerous when the fetus presents by the breech, even in the best possible circumstances. The greater likelihood of a low Apgar score might be due to an inherent abnormality which in itself had caused the breech presentation but perhaps is more likely to be due to respiratory efforts before delivery of the head. A mature, healthy fetus is probably well able to withstand this extra insult, but for a breech-presenting fetus which becomes distressed during preterm labour the added hazard might be important. If the hypothesis that delay in delivery of the fetal head causes the depression is correct then this is another reason to consider delivery by classical caesarean section in cases of breech presentation where the fetus is already compromised.
منابع مشابه
Effect of propranolol and metoprolol on parathyroid hormone and calcitonin secretions in uraemic patients.
Nine uraemic patients not being treated by dialysis received intravenous propranolol 1 microgram/kg/min for 85 minutes after a priming dose of 1 mg. Fifteen days later, six of them received intravenous metoprolol 1.2 microgram/kg/min after a priming dose of 1.2 mg. Plasma concentrations of parathyroid hormone (PTH) and calcitonin fell significantly after propranolol but not after metoprolol, wh...
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Secondary hyperparathyroidism classically appears during the course of chronic renal failure and sometimes after renal transplantation. Understanding the mechanisms by which parathyroid hormone (PTH) synthesis and secretion are normally regulated is important in devising methods to regulate overactivity and hyperplasia of the parathyroid gland after the onset of renal insufficiency. Rapid regul...
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concentration, which is recognized by a sevenIntroduction transmembrane-domain receptor with a large extracellular amino-terminal region, the calcium-sensing The development of secondary hyperparathyroidism receptor [4]. A decrease in extracellular calcium is a major risk factor for the development of renal concentration leads not only to an increase in PTH osteodystrophy. In cases of chronic r...
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Parathyroid hormone (PTH), which is elevated in patients with chronic renal failure, has been shown to participate in the development of vascular calcification. Previous studies have demonstrated that PTH may promote endothelial expressions of proinflammatory parameters. On the basis of these data, we evaluated whether PTH may have an impact on endothelial osteoprotegerin (OPG), a vascular-prot...
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عنوان ژورنال:
- British medical journal
دوره 281 6251 شماره
صفحات -
تاریخ انتشار 1980